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By Ernest Furchtgott

ISBN-10: 1441933115

ISBN-13: 9781441933119

ISBN-10: 1475744633

ISBN-13: 9781475744637

I first met Ernest Furchtgott twenty-five years in the past after becoming a member of the school of the varsity of Social paintings on the college of South Carolina. at the moment, Ernie chaired the dep. of Psychology. within the following 3 years we collaborated with a tutorial Committee on Gerontology in conceptualizing and shaping the University's certificates of Graduate examine in Gerontology application, guiding it to ultimate approval by means of the South Carolina fee on greater schooling. For two decades we crew­ taught our graduate-level direction, "Psychosocial methods to Geron­ tology," related to colleagues from comparable disciplines. through the years, we tested and together graded 1000s of study posters ready via our graduate scholars in gerontology as their ultimate direction requirement. numerous years in the past, Ernie officially retired from the collage. He in­ stantly agreed to my request that he proceed educating the psychology of getting older section of our interdisciplinary direction. On campus approximately on a daily basis when you consider that retirement, Ernie often telephoned to debate are­ cent article within the Gerontologist or a paper presentation that had ex­ pointed out him on the Gerontological Society's annual medical assembly. He maintained a transparent presence within the educational community.

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1990) as well as depression, which is usually more common in older individuals, may contribute to the higher levels of dex nonsuppression in more older than younger persons. Also, there seems to be some evidence that in approximately 50% of SDAT or multi-infarct patients, there is some dex resistance (Sapolsky & McEwen, 1988). It is plausible that in many studies in which older persons exhibited dexresistant depression, early stages of SDAT were present, but the latter are usually difficult to ascertain.

As we have previously indicated, numerous environmental factors, hospitalization, or drug usage, which frequently occur in a depressed population, will influence immune reactivity. Depression may be conceptualized as the consequence of ex- Biological Foundations 37 posure to a major social stressor, such as loss of a loved one or helplessness. Thus, the decrements in immune functions in individuals who have been exposed to stressors are not unexpected. Even the anticipation of stress may reduce T cells (Kemeny, Cohen, Zegans, & Conant, 1989).

A number of studies found no evidence that the menopause, with its dramatic drop in estrogens and progesterone, leads to any increases in clinical depression (Matthews, 1992; McKinlay, McKinlay, & Brambilla, 1987). We can conclude, therefore, that the gonadal hormone changes associated with aging in women have their major influence on the cardiovascular and musculoskeletal system. The commonly observed behavioral changes during the menopause are mainly the results of changes in other organismic systems affected by the gonadal hormones.

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Aging and Human Motivation by Ernest Furchtgott


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